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    A new discovery: The prevalence/incidence of different non-contagious diseases are associated with the antibiotic consumption patterns. DE

    A new discovery by Ternak, Gabor;

    The prevalence/incidence of different non-contagious diseases are associated with the antibiotic consumption patterns. DE

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      • Publisher's listprice EUR 84.90
      • The price is estimated because at the time of ordering we do not know what conversion rates will apply to HUF / product currency when the book arrives. In case HUF is weaker, the price increases slightly, in case HUF is stronger, the price goes lower slightly.

        36 014 Ft (34 299 Ft + 5% VAT)
      • Discount 5% (cc. 1 801 Ft off)
      • Discounted price 34 213 Ft (32 584 Ft + 5% VAT)

    36 014 Ft

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    Product details:

    • Publisher LAP Lambert Academic Publishing
    • Date of Publication 1 January 2022
    • Number of Volumes Großformatiges Paperback

    • ISBN 9786204984452
    • Binding Paperback
    • No. of pages224 pages
    • Size 16x152x222 mm
    • Weight 315 g
    • Language English
    • 387

    Categories

    Long description:

    The "growth promoting effect" of antibiotics was discovered over seventy years ago, and was utilized for enhancing meat production through mixing antibiotics to animal fodder. This effect had been observed in humans as well. Considerable scientific evidence has proved that antibiotics are capable of producing modifications of the intestinal microbiome (dysbiosis) and hence it can trigger inflammatory molecular cascades leading to different diseases.Hypothesis was developed that if different classes of antibiotics induce different alterations in the microbiome (dysbiosis), it might be associated with the occurrence of different metabolic (obesity, diabetes), neurodegenerative (Parkinson's disease, Alzheimer's disease, multiple sclerosis, etc.) and with certain malignant diseases, among others.This novel observation provides unified explanation for the pandemic-like appearance of the above diseases as different classes of antibiotics inducing different dysbiosis, which enhance or inhibit the development of those ailments through different mediator molecules via the gut-brain axis and other mechanisms.

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