
Translational Approaches to Unravelling Non-Motor Symptoms of Parkinson's disease
Series: International Review of Neurobiology; 174;
- Publisher's listprice EUR 150.00
-
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- Discount 20% (cc. 12 726 Ft off)
- Discounted price 50 904 Ft (48 480 Ft + 5% VAT)
63 630 Ft
Availability
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Product details:
- Publisher Academic Press
- Date of Publication 7 February 2024
- ISBN 9780323991834
- Binding Hardback
- No. of pages274 pages
- Size 228x152 mm
- Weight 560 g
- Language English 577
Categories
Long description:
Translational Approaches to Non-Motor Symptoms of Neurodegenerative Diseases, Volume 173 in the International Review on Neurobiology series, highlights new advances in the field with this new volume presenting interesting chapters on topics such as A general clinical overview of the non-motor symptoms, Stress/fatigue/cognitive decline Sleep/autonomic failure, A general clinical overview of the non-motor symptoms - psychiatric symptoms, Psychosis, Cognitive Decline, Non-clinical Aspect of non-motor symptoms: Animal Models, Assessment and treatment of cognitive decline in rodent and primate models, Pain in PD models, Affective disorders in PD models, Sleep and circadian dysregulation and EEG disruption in rodent models, and much more.
MoreTable of Contents:
A general clinical overview of the non-motor symptoms 1. General Clinical Overview 2. Stress/fatigue/ cognitive decline Sleep / autonomic failure A general clinical overview of the non-motor symptoms - psychiatric symptoms 3. Psychosis 4. Cognitive Decline Non-clinical Aspect of non-motor symptoms: Animal Models 5. Assessment and treatment of cognitive decline in rodent and primate models 6. Pain in PD models 7. Affective disorders in PD models 8. Sleep and circadian dysregulation and EEG disruption in rodent models of PD 9. Gut dysmotility in rodents and its central origin 10. Bladder dysfunction in rodent models of PD with a translational approach to bladder hyperreflexia in man 11. Long term adaptive changes to autonomically innervated smooth muscle contractility in a primate model of PD
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